Thromboembolien Lungenarterie links Thromboemboli and thromboembolisms. About Transcript. Clarifying difference between a thrombus and an embolus (and between thrombosis and embolism). Created by Sal Khan.


Thromboembolien Lungenarterie links


See all citations in PubMed. See citations in PubMed for homologs of this gene provided by HomoloGene. See variants in ClinVar. See studies and variants in dbVar. These reference sequences exist independently of genome builds. These reference sequences are curated independently Thromboembolien Lungenarterie links the genome annotation cycle, so their versions may not match the RefSeq versions in the current genome build.

Identify version mismatches by comparing the version of the RefSeq in this section to the one reported in Thromboembolien Lungenarterie links regions, transcripts, and products above. The following sections contain reference sequences that belong to a specific genome build. This section includes genomic Reference Sequences RefSeqs from all assemblies on which this gene is annotated, such as RefSeqs for chromosomes and scaffolds contigs from both reference and alternate assemblies.

Model RNAs and proteins are also reported here. The following LinkOut resources are supplied by external providers. These providers are learn more here for maintaining the links.

Turn recording back on. National Center for Biotechnology InformationU. Gene Genes and mapped phenotypes. Result Filters Gene sources Clear. Chromosome locations Select an organism by click at this page or scrolling Homo sapiens human.

Summary Go to the top of the page Help. This binding results in the activation of Thromboembolien Lungenarterie links C, which degrades clotting factors Va and VIIIa and reduces the http://zum-verklaerten-christus.de/betaliqeh/rosskastanie-therapeutische-eigenschaften-und-verwendung-bei-krampfadern.php of thrombin generated.

Thromboembolien Lungenarterie links in this gene are a cause Thromboembolien Lungenarterie links thromboembolic disease, also known as inherited thrombophilia.

Genomic context Go to the top of the page Help. Genomic regions, transcripts, and products Go to the top Thromboembolien Lungenarterie links the page Help.

Go Thromboembolien Lungenarterie links reference sequence details. Bibliography Go to the top of the page Help. Related articles in PubMed Association of thrombomodulin c. Hu B, et al. Gene, Sep PMID Failure to replicate thrombomodulin genetic variant predictors of venous thromboembolism in African Americans.

Folsom AR, et al. Blood, Aug 3. Biomed Res Int, Meyerson H, et al. Hum Pathol, Oct. PMID Whole exome sequencing identifies a mutation in thrombomodulin as the genetic cause of a Thromboembolien Lungenarterie links platelet disorder in a family with normal platelet function.

Maclachlan A, et al. Taken together, our findings suggest that TM-PKCdelta interaction may Thromboembolien Lungenarterie links to cardiovascular disorders by affecting monocye differentiation, which may develop future therapeutic applications. Whole exome sequencing identifies a mutation in thrombomodulin as the Thromboembolien Lungenarterie links cause of a suspected platelet disorder in a family with normal platelet function. Effects of alloantibodies to human leukocyte antigen on endothelial expression and serum levels of thrombomodulin.

Fibrinogen splice variation and cross-linking: Thrombomodulin promotes focal adhesion kinase activation and contributes to angiogenesis by binding to fibronectin.

Failure to replicate thrombomodulin genetic variant predictors of venous thromboembolism in African Americans. Phenotypes Go to the top of the page Help. Genetic Atypical Hemolytic-Uremic Syndrome. Variation Go to the top of the page Help. HIV-1 interactions Go to the top of the page Help. Pathways from BioSystems Go to the top of the page Help. AGE-RAGE signaling pathway in diabetic complications, organism-specific biosystem from KEGG AGE-RAGE signaling pathway in diabetic Thromboembolien Lungenarterie links, organism-specific biosystem Advanced glycation end products AGEs are a complex group of Thromboembolien Lungenarterie links produced through the non-enzymatic glycation and oxidation of proteins, lipids and nucleic acids, primarily due to aging and un AGE-RAGE signaling pathway in diabetic complications, conserved biosystem Advanced glycation end products AGEs are a complex group of compounds produced through the non-enzymatic glycation and oxidation of proteins, lipids click the following article nucleic acids, primarily due to aging and un Cell Thromboembolien Lungenarterie links interactions at the vascular wall, organism-specific biosystem Leukocyte extravasation is a rigorously controlled process that guides white cell movement from the vascular lumen to sites of tissue inflammation.

The powerful adhesive interactions that are require Common Pathway of Fibrin Clot Formation, organism-specific biosystem The common pathway consists of the cascade of activation events leading from the formation of activated factor X to the formation of active thrombin, the cleavage of fibrinogen by thrombin, and the f Complement and Coagulation Cascades, organism-specific biosystem Thromboembolien Lungenarterie links coagulation is a series of coordinated and link proenzyme-to-serine protease conversions likely to be localized on the surfaces learn more here activated cells in vivo.

It culminates in the for Complement and coagulation cascades, organism-specific biosystem The this web page system is a proteolytic cascade in blood plasma and a mediator of innate immunity, a Thromboembolien Lungenarterie links defense mechanism against pathogens.

There are three pathways of complement activation Complement and coagulation cascades, conserved biosystem The complement system is a proteolytic cascade in blood plasma and a mediator of innate immunity, a nonspecific Thromboembolien Lungenarterie links mechanism against pathogens. Fluid shear stress and atherosclerosis, organism-specific biosystem Shear stress represents the frictional force that the flow of Thromboembolien Lungenarterie links exerts at the endothelial surface of the vessel wall and plays a central role in vascular biology and contributes to the progress o Fluid shear stress and atherosclerosis, conserved biosystem Shear stress represents the frictional force that the flow of blood exerts at the endothelial surface of the vessel wall and plays a central role in Thromboembolien Lungenarterie links biology and contributes to the progress o Formation of Fibrin Clot Clotting Cascadeorganism-specific biosystem The formation of a fibrin clot at the site of an injury to the wall of a normal blood vessel is an essential part of the process to stop blood loss after vascular injury.

The reactions Thromboembolien Lungenarterie links lead to Hemostasis, organism-specific biosystem Hemostasis is a physiological response that culminates in the arrest of bleeding from an injured vessel. Under normal conditions the vascular endothelium supports vasodilation, inhibits platelet adhe Validated transcriptional targets of AP1 family members Fra1 and Fra2, organism-specific biosystem Validated transcriptional targets of AP1 family members Fra1 and Fra2.

Interactions Go to the top of the page Help. General gene information Go Thromboembolien Lungenarterie links the top of the page Thromboembolien Lungenarterie links. Orthologs from Thromboembolien Lungenarterie links Pipeline: Inferred from Thromboembolien Lungenarterie links Interaction more info.

Traceable Author Statement more info. Inferred from Thromboembolien Lungenarterie links Annotation more info. Inferred from Direct Assay more info. General protein information Go to the top more info the page Thromboembolien Lungenarterie links. Preferred Names thrombomodulin Names CD antigen fetomodulin.

Thromboembolien Lungenarterie links maintained independently of Annotated Genomes These reference sequences exist independently of genome builds. Explain These reference sequences are curated independently of the genome annotation cycle, so their versions may not match the RefSeq versions in the current genome build.

Explain This section includes genomic Reference Sequences RefSeqs from all assemblies on which this gene is annotated, such as RefSeqs for chromosomes and scaffolds Thromboembolien Lungenarterie links from both reference and alternate assemblies.

Related sequences Go to the top of Thromboembolien Lungenarterie links page Help. Additional links Thromboembolien Lungenarterie links to the top of the page Help. BioAssays related to the gene by protein target or RNAi target. Summarized PubChem Data on the gene, showing the active data by default.

BioAssays that contain the gene as the target of a RNAi reagent, which is identified Thromboembolien Lungenarterie links a hit in a RNAi screening and flagged as "active" in the corresponding BioAssay record. BioAssays that contain the gene as the target of a RNAi reagent. Full text in PubMedCentral identified from shared sequence links. Overlapping genes and two nearest non-overlapping genes on either side.

Links to other resources. Clear Turn Off Thromboembolien Lungenarterie links On. Please review our privacy policy. Atypical hemolytic-uremic syndrome 6 MedGen: Thrombophilia due to thrombomodulin defect MedGen: Envelope transmembrane glycoprotein gp


Thromboembolien Lungenarterie links Unsupported Transport Layer Security Protocol

In Abhängigkeit von der Lokalisation des embolischen Prozesses werden Thromboembolien Lungenarterie links klinisch-anatomische Varianten der Lungenembolie PE Thromboembolien Lungenarterie links. Schwere klinische Symptome sind mit schweren Beeinträchtigungen der Thromboembolien Lungenarterie links Kollaps, akutes Atemversagen, Atemstillstand, oft Kammerflimmern verbunden, die Erkrankung verläuft katastrophal schnell und führt in wenigen Minuten zum Tod.

Infarkte der Lungen Thromboembolien Lungenarterie links in diesen Fällen in der Thromboembolien Lungenarterie links nicht erkannt haben keine Zeit sich zu entwickeln. Es ist gekennzeichnet durch einen plötzlichen Beginn und eine rasch fortschreitende Entwicklung der Symptome der respiratorischen, kardiovaskulären und zerebralen Insuffizienz.

Die meisten please click for source ihnen im klinischen Bild werden von der oben genannten klinischen Syndrome mit ihrer extremen Schwere dominiert.

Das klinische Bild ist weniger dramatisch. Die am häufigsten kombinierten: Schmerzen in Thromboembolien Lungenarterie links Brust sind mit Schmerzen im rechten oberen Quadranten kombiniert.

Klinische Manifestationen dauern mehrere Tage. Die Bedeutung dieser Symptome erhöht, wenn sie bei Patienten mit kongestiver Herzinsuffizienz, bösartigen Tumoren nach der Operation, Knochenbrüchen beobachtet Thromboembolien Lungenarterie links, nach der Lieferung Schlaganfällen, die Detektionseigenschaften Phlebothrombosen. Vorgeschlagen, eine Klassifizierung Thromboembolien der Pulmonalarterie Lungenemboliedie die Lokalisation von Läsionen berücksichtigt, die den Grad der Lungenperfusion Läsion Volumender Schwere der hämodynamischen Störungen und Thromboembolien Lungenarterie links der Krankheit Störungen Prognose und Behandlung verursachen.

Ein neuer Faktor, der die Entwicklung von Myokardinfarkt beeinflusst. Kamille zur Mundspülung mit Halsschmerzen und Entzündungen. Rezepte für Diät auf Schlankheitskohl. Körperliche Übungen helfen, das Denken schnell zu klären. Diät auf dem Kohl: Thromboembolie der Thromboembolien Lungenarterie links PE - Klassifikation.

In Abhängigkeit von der Lokalisation des embolischen Prozesses werden folgende klinisch-anatomische Varianten der Lungenembolie PE unterschieden: Der akuteste "fulminante" Verlauf wird beobachtet, wenn die Embolie des Hauptstammes oder beider Hauptäste der Lungenarterie durch den Embolus vollständig blockiert ist.

Die Entwicklung eines Lungeninfarktes für diese Variante ist nicht typisch, tritt aber auf. Die Krankheit dauert ein bis mehrere Wochen. Die akuten Erscheinungsformen der Anfangsperiode schwächen sich etwas ab, die Erkrankung nimmt die Thromboembolien Lungenarterie links Art mit der Erhöhung der rechtsventrikulären und respiratorischen Mangelhaftigkeit langsam an.

Vor diesem Thromboembolien Lungenarterie links kann es wiederholte embolische Episoden geben, die durch Verschlimmerung der Symptome oder das Auftreten von Anzeichen eines Lungeninfarkts gekennzeichnet sind. Oft kommt Thromboembolien Lungenarterie links zu einem tödlichen Ausgang - plötzlich durch wiederholte Embolie des Hauptstammes oder der Hauptäste oder durch fortschreitende Herz-Lungen-Insuffizienz. Eine rezidivierende PE tritt häufiger auf dem Hintergrund von kardiovaskulären Visit web page, malignen Neoplasmen, nach Operationen an den Organen der Bauchhöhle auf.

Klassifizierung von PE Yu. Andere Artikel zum Thema. Die jährliche Inzidenz von tiefen Venenthrombosen des Unterschenkels beträgt pro Einwohner. Thromboembolie der Lungenarterie PE - Symptome. Die charakteristischste subjektive Manifestation der Krankheit ist der plötzliche Schmerz hinter dem Brustbein verschiedener Natur. Thromboembolie der Lungenarterie PE - Diagnose. Das plötzliche Auftreten der oben genannten Krankheitsbilder: In sozialen Just click for source teilen.

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Heart disease and heart attacks

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Trotzdem stellt eine Lungenarterie- dien, die das Risiko venöser Thromboembolien bei schwangeren Frauen mit Thrombophilie untersuch-ten, zeigte.
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Thromboemboli and thromboembolisms. About Transcript. Clarifying difference between a thrombus and an embolus (and between thrombosis and embolism). Created by Sal Khan.
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Thrombomodulin mediates the migration of cervical cancer cells through the regulation of epithelial-mesenchymal transition biomarkers. Data indicate that thrombomodulin (TM) expression may regulate cell growth and migration in ovarian cancer cells, and suggest that TM may be a novel prognostic and therapeutic target for ovarian cancer.
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